overperfusion syndrome
过度灌注综合征
cerebral overperfusion
脑过度灌注
overperfusion injury
过度灌注损伤
tissue overperfusion
组织过度灌注
pulmonary overperfusion
肺过度灌注
prevent overperfusion
预防过度灌注
renal overperfusion
肾过度灌注
overperfusion causes
过度灌注导致
overperfusion of
过度灌注
treating overperfusion
治疗过度灌注
cerebral overperfusion syndrome can occur after carotid endarterectomy in patients with compromised autoregulation.
颈动脉内膜切除术后，脑灌注过度综合征可能发生在自我调节功能受损的患者身上。
tissue overperfusion leads to edema and cellular damage due to excessive oxygen delivery beyond metabolic needs.
组织灌注过度导致水肿和细胞损伤，因为氧气输送超过了代谢需求。
the physician monitored for signs of organ overperfusion during the aggressive fluid resuscitation protocol.
医生在积极的液体复苏方案中监测器官灌注过度的迹象。
capillary overperfusion causes increased hydrostatic pressure that promotes plasma extravasation into interstitial spaces.
毛细血管灌注过度导致静水压增加，促进血浆渗出到组织间隙。
regional overperfusion may explain the preferential distribution of contrast enhancement in certain vascular territories.
局部灌注过度可以解释某些血管区域造影剂增强的优先分布。
overperfusion injury to the brainstem resulted from the rapid correction of chronic cerebral hypoperfusion.
脑干的灌注过度损伤是由于慢性脑灌注不足的快速纠正造成的。
the cardiology team adjusted the inotropic support to prevent myocardial overperfusion and subsequent reperfusion injury.
心脏病学团队调整了正性肌力支持以防止心肌灌注过度和随后的再灌注损伤。
post-operative monitoring revealed pulmonary overperfusion that contributed to the development of pulmonary edema.
术后监测显示肺灌注过度促成了肺水肿的发展。
hepatic overperfusion syndrome was observed following successful revascularization of the previously ischemic liver.
在先前缺血的肝脏成功血运重建后，观察到肝脏灌注过度综合征。
the patient developed renal overperfusion after aggressive volume expansion, necessitating diuretic therapy.
患者在积极的容量扩张后出现肾灌注过度，需要利尿治疗。
risk factors for cerebral overperfusion include severe carotid stenosis and inadequate collateral circulation.
脑灌注过度的危险因素包括严重的颈动脉狭窄和侧支循环不足。
pulmonary overperfusion can occur in conditions where left ventricular output suddenly increases.
肺灌注过度可能发生在左心室输出量突然增加的情况下。
overperfusion syndrome
过度灌注综合征
cerebral overperfusion
脑过度灌注
overperfusion injury
过度灌注损伤
tissue overperfusion
组织过度灌注
pulmonary overperfusion
肺过度灌注
prevent overperfusion
预防过度灌注
renal overperfusion
肾过度灌注
overperfusion causes
过度灌注导致
overperfusion of
过度灌注
treating overperfusion
治疗过度灌注
cerebral overperfusion syndrome can occur after carotid endarterectomy in patients with compromised autoregulation.
颈动脉内膜切除术后，脑灌注过度综合征可能发生在自我调节功能受损的患者身上。
tissue overperfusion leads to edema and cellular damage due to excessive oxygen delivery beyond metabolic needs.
组织灌注过度导致水肿和细胞损伤，因为氧气输送超过了代谢需求。
the physician monitored for signs of organ overperfusion during the aggressive fluid resuscitation protocol.
医生在积极的液体复苏方案中监测器官灌注过度的迹象。
capillary overperfusion causes increased hydrostatic pressure that promotes plasma extravasation into interstitial spaces.
毛细血管灌注过度导致静水压增加，促进血浆渗出到组织间隙。
regional overperfusion may explain the preferential distribution of contrast enhancement in certain vascular territories.
局部灌注过度可以解释某些血管区域造影剂增强的优先分布。
overperfusion injury to the brainstem resulted from the rapid correction of chronic cerebral hypoperfusion.
脑干的灌注过度损伤是由于慢性脑灌注不足的快速纠正造成的。
the cardiology team adjusted the inotropic support to prevent myocardial overperfusion and subsequent reperfusion injury.
心脏病学团队调整了正性肌力支持以防止心肌灌注过度和随后的再灌注损伤。
post-operative monitoring revealed pulmonary overperfusion that contributed to the development of pulmonary edema.
术后监测显示肺灌注过度促成了肺水肿的发展。
hepatic overperfusion syndrome was observed following successful revascularization of the previously ischemic liver.
在先前缺血的肝脏成功血运重建后，观察到肝脏灌注过度综合征。
the patient developed renal overperfusion after aggressive volume expansion, necessitating diuretic therapy.
患者在积极的容量扩张后出现肾灌注过度，需要利尿治疗。
risk factors for cerebral overperfusion include severe carotid stenosis and inadequate collateral circulation.
脑灌注过度的危险因素包括严重的颈动脉狭窄和侧支循环不足。
pulmonary overperfusion can occur in conditions where left ventricular output suddenly increases.
肺灌注过度可能发生在左心室输出量突然增加的情况下。
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