ltc4 system
ltc4系统
ltc4 error
ltc4错误
ltc4 code
ltc4代码
ltc4 module
ltc4模块
ltc4 protocol
ltc4协议
ltc4 signal
ltc4信号
ltc4 function
ltc4功能
ltc4 interface
ltc4接口
ltc4 component
ltc4组件
ltc4 design
ltc4设计
the ltc4 pathway is activated during allergic inflammation, leading to bronchoconstriction.
白三烯c4通路在过敏性炎症期间被激活，导致支气管收缩。
inhibiting ltc4 synthesis reduces airway hyperresponsiveness in asthmatic models.
抑制白三烯c4合成可降低哮喘模型的气道高反应性。
the ltc4 receptor antagonists block smooth muscle contraction and mucus secretion.
白三烯c4受体拮抗剂阻断平滑肌收缩和黏液分泌。
measuring ltc4 levels in exhaled breath condensate helps monitor asthma severity.
测量呼气冷凝液中的白三烯c4水平有助于监测哮喘严重程度。
ltc4 activity is enhanced by oxidative stress in endothelial cells.
白三烯c4活性在内皮细胞中受到氧化应激的增强。
the ltc4 formation requires the enzyme leukotriene c4 synthase.
白三烯c4的生成需要白三烯c4合酶。
pharmacological inhibition of ltc4 release attenuates vascular leakage.
药物抑制白三烯c4释放可减轻血管渗漏。
the involvement of ltc4 in inflammatory responses has been documented in numerous studies.
白三烯c4在炎症反应中的作用已在众多研究中得到证实。
targeting ltc4 signaling pathways may provide novel therapeutic strategies for allergic diseases.
靶向白三烯c4信号通路可能为过敏性疾病提供新型治疗策略。
ltc4 mediated inflammation can be suppressed by corticosteroids in clinical settings.
白三烯c4介导的炎症在临床环境中可被皮质类固醇抑制。
the ltc4 dependent chemotaxis of eosinophils contributes to airway remodeling.
白三烯c4依赖性嗜酸粒细胞趋化促进气道重塑。
combining ltc4 inhibitors with beta-agonists improves lung function in copd patients.
联合使用白三烯c4抑制剂与贝塔受体激动剂可改善慢性阻塞性肺疾病患者的肺功能。
ltc4 system
ltc4系统
ltc4 error
ltc4错误
ltc4 code
ltc4代码
ltc4 module
ltc4模块
ltc4 protocol
ltc4协议
ltc4 signal
ltc4信号
ltc4 function
ltc4功能
ltc4 interface
ltc4接口
ltc4 component
ltc4组件
ltc4 design
ltc4设计
the ltc4 pathway is activated during allergic inflammation, leading to bronchoconstriction.
白三烯c4通路在过敏性炎症期间被激活，导致支气管收缩。
inhibiting ltc4 synthesis reduces airway hyperresponsiveness in asthmatic models.
抑制白三烯c4合成可降低哮喘模型的气道高反应性。
the ltc4 receptor antagonists block smooth muscle contraction and mucus secretion.
白三烯c4受体拮抗剂阻断平滑肌收缩和黏液分泌。
measuring ltc4 levels in exhaled breath condensate helps monitor asthma severity.
测量呼气冷凝液中的白三烯c4水平有助于监测哮喘严重程度。
ltc4 activity is enhanced by oxidative stress in endothelial cells.
白三烯c4活性在内皮细胞中受到氧化应激的增强。
the ltc4 formation requires the enzyme leukotriene c4 synthase.
白三烯c4的生成需要白三烯c4合酶。
pharmacological inhibition of ltc4 release attenuates vascular leakage.
药物抑制白三烯c4释放可减轻血管渗漏。
the involvement of ltc4 in inflammatory responses has been documented in numerous studies.
白三烯c4在炎症反应中的作用已在众多研究中得到证实。
targeting ltc4 signaling pathways may provide novel therapeutic strategies for allergic diseases.
靶向白三烯c4信号通路可能为过敏性疾病提供新型治疗策略。
ltc4 mediated inflammation can be suppressed by corticosteroids in clinical settings.
白三烯c4介导的炎症在临床环境中可被皮质类固醇抑制。
the ltc4 dependent chemotaxis of eosinophils contributes to airway remodeling.
白三烯c4依赖性嗜酸粒细胞趋化促进气道重塑。
combining ltc4 inhibitors with beta-agonists improves lung function in copd patients.
联合使用白三烯c4抑制剂与贝塔受体激动剂可改善慢性阻塞性肺疾病患者的肺功能。
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