kaliuresis occurs
发生钾尿症
excessive kaliuresis
过度钾尿症
kaliuresis develops
钾尿症发展
kaliuresis indicates
钾尿症表明
treating kaliuresis
治疗钾尿症
kaliuresis causes
钾尿症引起
severe kaliuresis
严重钾尿症
kaliuresis results
钾尿症导致
preventing kaliuresis
预防钾尿症
abnormal kaliuresis
异常钾尿症
the patient exhibited increased kaliuresis following diuretic therapy.
该患者在利尿剂治疗后出现尿钾排泄增加。
excessive kaliuresis can lead to dangerous hypokalemia.
过度尿钾排泄可能导致危险的低钾血症。
the drug effectively promotes kaliuresis in patients with fluid overload.
该药物有效促进液体超负荷患者的尿钾排泄。
renal kaliuresis was significantly elevated during the experimental period.
实验期间肾脏尿钾排泄显著增加。
chronic hypertension often correlates with abnormal kaliuresis patterns.
慢性高血压通常与异常的尿钾排泄模式相关。
the doctor monitored the patient's kaliuresis rate closely.
医生密切监测患者的尿钾排泄率。
loop diuretics typically enhance kaliuresis more than thiazides.
袢利尿剂通常比噻嗪类更能增强尿钾排泄。
aldosterone deficiency reduces kaliuresis and causes potassium retention.
醛固酮缺乏会减少尿钾排泄并导致钾潴留。
the kaliuresis test revealed impaired potassium excretion.
尿钾排泄测试显示钾排泄受损。
metabolic alkalosis may stimulate kaliuresis through hormonal mechanisms.
代谢性碱中毒可能通过激素机制刺激尿钾排泄。
dietary potassium restriction can attenuate excessive kaliuresis.
限制饮食钾可以减轻过度的尿钾排泄。
the study measured 24-hour kaliuresis in all participants.
研究测量了所有参与者的24小时尿钾排泄量。
abnormal kaliuresis serves as an important diagnostic marker for renal tubular disorders.
异常尿钾排泄是肾小管疾病的重要诊断标志物。
kaliuresis occurs
发生钾尿症
excessive kaliuresis
过度钾尿症
kaliuresis develops
钾尿症发展
kaliuresis indicates
钾尿症表明
treating kaliuresis
治疗钾尿症
kaliuresis causes
钾尿症引起
severe kaliuresis
严重钾尿症
kaliuresis results
钾尿症导致
preventing kaliuresis
预防钾尿症
abnormal kaliuresis
异常钾尿症
the patient exhibited increased kaliuresis following diuretic therapy.
该患者在利尿剂治疗后出现尿钾排泄增加。
excessive kaliuresis can lead to dangerous hypokalemia.
过度尿钾排泄可能导致危险的低钾血症。
the drug effectively promotes kaliuresis in patients with fluid overload.
该药物有效促进液体超负荷患者的尿钾排泄。
renal kaliuresis was significantly elevated during the experimental period.
实验期间肾脏尿钾排泄显著增加。
chronic hypertension often correlates with abnormal kaliuresis patterns.
慢性高血压通常与异常的尿钾排泄模式相关。
the doctor monitored the patient's kaliuresis rate closely.
医生密切监测患者的尿钾排泄率。
loop diuretics typically enhance kaliuresis more than thiazides.
袢利尿剂通常比噻嗪类更能增强尿钾排泄。
aldosterone deficiency reduces kaliuresis and causes potassium retention.
醛固酮缺乏会减少尿钾排泄并导致钾潴留。
the kaliuresis test revealed impaired potassium excretion.
尿钾排泄测试显示钾排泄受损。
metabolic alkalosis may stimulate kaliuresis through hormonal mechanisms.
代谢性碱中毒可能通过激素机制刺激尿钾排泄。
dietary potassium restriction can attenuate excessive kaliuresis.
限制饮食钾可以减轻过度的尿钾排泄。
the study measured 24-hour kaliuresis in all participants.
研究测量了所有参与者的24小时尿钾排泄量。
abnormal kaliuresis serves as an important diagnostic marker for renal tubular disorders.
异常尿钾排泄是肾小管疾病的重要诊断标志物。
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